Hyperintense Signal Abnormality in Subarachnoid Spaces and Basal Cisterns on MR Images of Children Anesthetized with Propofol: New Fluid-attenuated Inversion Recovery Finding
Christopher G. Filippi
,a,
Aziz M. Ulu
a,
Doris Lina,
Linda A. Heiera and
Robert D. Zimmermana
a From the Department of Radiology, New York Presbyterian Hospital-Weill Medical College of Cornell University, New York, NY.
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FIG 1. MR images of a child with left middle cerebral artery ischemia and sickle cell disease.
A, Transaxial FLAIR (10002/172/1) image obtained with the child under propofol anesthesia has hyperintense signal abnormality in the basal cisterns.
B, Similar abnormal hyperintense signal is seen in the subarachnoid spaces bilaterally in the convexity region of this same patient.
C, Follow-up transaxial FLAIR (10002/172/1) image shows resolution of the signal abnormality in the basal cisterns. Chloral hydrate was administered, and patient's retainer was not removed, creating the paranasal sinus artifact that was not present on the previous study.
D, Resolution of the abnormal signal intensity in the convexity subarachnoid spaces is also noted on the follow-up image obtained with the child under chloral hydrate sedation.
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FIG 2. T1 value of chloral hydrate, propofol, and CSF determined in vitro at room temperature using an inversion recovery pulse sequence at 1.5 T. T1 value of CSF is 2.32 s, of propofol is 1.86 s, and of chloral hydrate is 0.2 s. The null time of CSF is 1.6 s, of propofol is 1.3 s, and of chloral hydrate is 0.14 s
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