Frequency of Subclinical Lacunar Infarcts in Ischemic Leukoaraiosis and Cerebral Autosomal Dominant Arteriopathy with Subcortical Infarcts and Leukoencephalopathy
Michael O’Sullivana,
Philip M. Richb,
Thomas R. Barricka,
Christopher A. Clarka and
Hugh S. Markusa
a Division of Clinical Neuroscience, St. George’s Hospital Medical School, London, England
b Department of Neuroradiology, St. George’s and Atkinson Morley’s Hospitals, London, England
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FIG 1. Examples of recent ischemic lesions and T2 shine-through effect. For recent lesions, high signal intensity is seen on the diffusion-weighted images (left), and corresponding reductions of diffusivity are seen on the ADC maps (right, arrows).
A, A recent asymptomatic ischemic lesions is seen in the left centrum semiovale in a patient with ischemic leukoaraiosis.
B, A recent asymptomatic ischemic lesions is seen in the left cerebellar hemisphere in a patient with CADASIL.
C, Two areas of hyperintensity are seen on the diffusion-weighted images of a different patient (left) and are found to correspond with increased ADC values (right, arrows), indicating that these are chronic lesions.
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