Early Uncoupling of Cerebral Blood Flow and Metabolism after Bilateral Thalamic Infarction
Beau M. Ancesa,
David S. Liebeskinda,b,
Andrew B. Newbergb,
Dina A. Jacobsa and
Abass Alavib
a Department of Neurology, University of Pennsylvania, Philadelphia, PA
b Department of Radiology, University of Pennsylvania, Philadelphia, PA

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FIG 1. Noncontrast CT on admission reveals a chronic left frontal infarct without acute ischemic changes.
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FIG 2. Emergent angiogram of the basilar artery (anteroposterior view) demonstrates absence of arterial occlusion.
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FIG 3. Subacute bilateral paramedian thalamic infarction depicted on noncontrast CT.
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FIG 4. Concomitant SPECT (A) and FDG-PET (B) during the subacute phase illustrates thalamic luxury perfusion and cortical diaschisis.
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FIG 5. At 6 months from stroke onset, concomitant SPECT (A) and FDG-PET (B) detail normal thalamic coupling and resolution of diffuse cortical diaschisis.
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