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Susac Syndrome: Report of Four Cases and Review of the Literature

Tai Hung Doa, Christiane Fischa and François Evoya

a From the Department of Radiology, CHUS—Hôpital Fleurimont, Sherbrooke, Quebec, Canada



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  		FIG 1. Case 1.

A, Axial T2-weighted MR image shows hyperintense lesions in the periventricular white matter and corpus callosum.

B, Axial T2-weighted MR image shows a small, hyperintense foci in the left posterior arm of the internal capsule.



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  		FIG 2. Case 2.

A, Axial T1-weighted postcontrast MR image shows leptomeningeal enhancement in the interpeduncular area.

B, Axial T2-weighted MR image shows hyperintense lesions in the corpus callosum and periventricular white matter.

C, Histopathologic examination shows areas of capillary hyaline thrombi with perivascular inflammatory cells (hematoxylin and eosin stain; magnification x400).

D, Retinal fluorescein angiography shows peripheral retinal arteriolar branch occlusions in the left eye.

E, Axial T2-weighted MR image shows bilateral hyperintense lesions in the thalamus and posterior arm of the internal capsule.

F, Axial T1-weighted postcontrast MR imaging shows enhancing lesions in the corpus callosum and the posterior arm of the internal capsule.



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  		FIG 3. Case 3.

A, Axial T1-weighted postcontrast MR image shows numerous foci of enhancement in the cerebellum.

B, Axial T2-weighted MR image shows small, hyperintense lesions in the right periventricular frontal white matter.

C, Audiogram shows moderate to severe sensorineural hearing loss on the left ear.



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  		FIG 4. Case 4.

A, Axial T2-weighted MR image shows hyperintense lesions in the left middle cerebellar peduncle.

B, Retinal fluorescein angiogram shows an inflamed left retinal arteriole with a leaking arteriolar wall.

C, Axial T2-weighted MR image shows increase number of hyperintense lesions in the cerebellum.





Copyright © 2008 by the American Society of Neuroradiology. Print ISSN: 0195-6108 Online ISSN: 1936-959X