Involvement of the Anterior Lobe of the Cerebellar Vermis in Perinatal Profound Hypoxia
D.J.A. Connollya,
E. Widjajab and
P.D. Griffithsc
a Department of Radiology, Sheffield Children’s Hospital, Sheffield, England
b Department of Radiology, Royal Hallamshire Hospital, Sheffield, England
c Academic Unit of Radiology, University of Sheffield, Sheffield, England
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Fig 1. Five-year-old girl with athetoid spastic quadriplegia.
A, Axial T2. Mild signal intensity change in the perirolandic white matter.
B, Axial T2. Typical signal intensity change in the posterior putamen and the ventrolateral thalamus bilaterally.
C, Axial T2. Typical signal intensity change in the anterior lobe of the vermis.
D, Axial T1. Low T1 signal intensity change in the vermis.
E, Axial FLAIR. High T2 signal intensity and cystic change.
F, Coronal T2. Anterior lobe of vermis high T2 signal intensity change.
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Fig 2. Nine-month-old girl with dyskinetic cerebral palsy.
A, Coronal T2. Central lobule of anterior lobe of the vermis high T2 signal intensity.
B, Axial FLAIR. High T2 signal intensity and cystic change of anterior lobe of vermis.
C, Sagittal T2. Central lobule of anterior lobe of vermis high T2 signal intensity.
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Fig 3. Seven-year-old boy with severe spastic quadriplegia.
A, Axial T2. Severe signal intensity change and volume loss in the perirolandic white matter.
B, Axial T2. Severe putaminal and thalamic volume loss and high T2 signal intensity bilaterally.
C, Axial T2. Mild vermian high T2 signal intensity change.
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