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Hyperintensity of the Precentral Gyral Subcortical White Matter and Hypointensity of the Precentral Gyrus on Fluid-Attenuated Inversion Recovery: Variation with Age and Implications for the Diagnosis of Amyotrophic Lateral Sclerosis

S. Ngaia, Y.M. Tanga, L. Dua and S. Stuckeya

a From the Department of Radiology, Princess Alexandra Hospital, Brisbane, Queensland, Australia


Figure 1
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  		Fig 1. A, 65-year-old patient. Axial FLAIR. There is subcortical white matter hyperintensity (arrows) that is isointense to the caudate nucleus, classified as "mildly hyperintense" and graded as "1.

B, Axial FLAIR. Signal intensities at the caudate nucleus and insular cortex in the same patient with grade 1 SWM hyperintensity.


Figure 2
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  		Fig 2. A, 76-year-old patient. Axial FLAIR. There is subcortical white matter hyperintensity (arrows) which is isointense to the insular cortex, classified as "distinctively hyperintense" and graded as "2."

B, Axial FLAIR. Signal intensities at the caudate nucleus and insular cortex in the same patient with grade 2 SWM hyperintensity.


Figure 3
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  		Fig 3. 61-year-old patient. Axial FLAIR. There is absence of subcortical white matter hyperintensity.


Figure 4
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  		Fig 4. 65-year-old patient. Axial FLAIR. There is a hypointense line in the precentral gyrus gray matter (arrows), designated "1."


Figure 5
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  		Fig 5. 61-year-old patient. Axial FLAIR. There is absence of a hypointense line in the precentral gyrus gray matter, designated "0."


Figure 6
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  		Fig 6. Percentage of subjects identified with SWM hyperintensity in different age groups (average of reader A and B).


Figure 7
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  		Fig 7. Percentage of subjects identified with PGGM hypointensity in different age groups (average of reader A and B).





Copyright © 2008 by the American Society of Neuroradiology. Print ISSN: 0195-6108 Online ISSN: 1936-959X