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AJNR - American Journal of Neuroradiology

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Wyburn-Mason Syndrome Associated with Thyroid Arteriovenous Malformation: A First Case Report

A.W. Lee^a, C.S. Chen^b, P. Gailloud^c and P. Nyquist^a

^a Departments of Cerebrovascular Neurology, Department of Radiology, Johns Hopkins Medical Institutes, Baltimore, Md
^b Neuro-Ophthalmology, Department of Radiology, Johns Hopkins Medical Institutes, Baltimore, Md
^c Division of Neuroradiology, Department of Radiology, Johns Hopkins Medical Institutes, Baltimore, Md

View larger version (143K): [in this window] [in a new window]   Fig 1. DSA of the left internal carotid artery, lateral view. There is an abnormal blush within the apex of the left orbit (O) and a left intracerebral (I) AVM extending posteriorly along the left optic nerve, tract, and radiations. This is fed by distal branches from the left internal carotid artery, as well as the anterior cerebral artery, a prominent anterior choroidal artery, and thalamic perforators from the posterior communicating artery, not demonstrated in the current view.

View larger version (140K): [in this window] [in a new window]   Fig 2. Lateral view of a left vertebral artery injection (V) showing the same left intracerebral AVM (A), as seen in Fig 1, fed by thalamic perforators arising from the left posterior communicating artery.

View larger version (146K): [in this window] [in a new window]   Fig 3. DSA of the right external carotid artery injection. A, Early arterial phase, lateral view, showing 2 nodular areas of abnormal blush fed by prominent arterial branches in the thyroid gland. B, Oblique view, midarterial phase, showing increase in prominence of the thyroid gland arteriovenous malformation and early draining veins (not shown here). These findings are consistent with multiple high-flow vascular malformations.