Article Figures & Data
Figures
- Fig 1.
DWI studies in patients with HE show abnormal signals at the bilateral temporo-occipital lobes, patient 1 (A); bilateral precentral gyrus, patient 2 (B); and bilateral internal capsules, patient 3 (C).
Tables
Summary of clinical features and lesion distributions on DWI in patients with hypoglycemic encephalopathy
No. Age (yr) Sex SBP (mm Hg) Body Temp (°C) GCS Glucose (mg/dL) Ca (mg/dL) Na (mg/dL) Cr (mg/dL) EEG Hypoglycemia-DWI Interval (hr) DWI Lesions Outcome 1 76 F 155 39.4 5 36 10 137 0.7 Diffuse slow waves, 2–7 Hz 48 Bilateral internal capsules, centrum semiovale, precentral gyrus, and temporo-parieto-occipital regions Coma 2 73 F 141 37.5 9 24 9.6 134 1.28 Diffuse slow waves, 2–5 Hz 36 Bilateral precentral gyrus Recovered 3 76 M 116 36.2 5 37 8.7 135 0.93 ND 6 Bilateral internal capsules, precentral gyrus Recovered 4 47 F 129 35.9 7 41 9.7 136 0.7 Normal 48 – Recovered 5 83 M 186 37.0 8 49 6 141 0.99 Diffuse slow waves, 3–6 Hz 55 – Recovered 6 78 F 147 36.8 7 25 5 132 4.1 ND 24 – Coma 7 64 F 161 34.3 6 37 7.8 138 2.22 Diffuse slow waves, 2–5 Hz 8 – Recovered 8 57 M 81 36.2 9 39 5.2 142 3.59 Diffuse slow waves, 3–4 Hz 120 – Recovered 9 78 M 127 36.9 10 35 8.6 137 0.65 Diffuse slow waves, 4–7 Hz 9 – Recovered -
Note:—Ca indicates calcium; Cr, creatinine; Na, sodium; ND, not performed; Temp, temperature; SBP, systolic blood pressure; –, no DWI lesion.
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Serum Calcium Concentration Affects Signal Changes on Diffusion-Weighted Imaging in Hypoglycemic Encephalopathy
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