Demographic information and summary of imaging findings for 6 subjects with pAgCC

SubjectAge (yr)SexStructural Malformations*Fractional Fragment PositionPredicted Homotopic ConnectionsObserved Homotopic ConnectionsObserved Heterotopic Connections§
111FNone0.32–0.45PFAF, PF, OTNone
248FRight lateral periventricular nodular heterotopia0.09–0.44AF, PFAF, PF, OTNone
336MLeft frontal periventricular nodular heterotopia0.10–0.33AF, PFAFAF(L)-OT(R)
437FNone0.46–0.70PFPF, OTPF(L)-OT(R)
570MNone0.38–0.50PFAF, OTAF(L)-OT(R)
637MNone0.20–0.83PF, PAAF, PF, OTAF(R)-OT(L)
  • Note:—AF indicates anterior frontal; PF, posterior frontal; PA, parietal; OT, occipitotemporal; L, left; R, right; pAgCC, partial agenesis of the corpus callosum.

  • * Structural malformations and fragment position (as a fraction of normal callosal length) were determined from high-resolution T1-weighted structural images.

  • Predicted homotopic connectivity is based on fragment position.

  • Observed homotopic and heterotopic callosal connections are determined from QBI tractography.

  • § For heterotopic connections, the hemisphere of each connected cortical region is also denoted in parentheses.