Original articleMRI findings in children infected by Borrelia burgdorferi☆,☆☆
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2021, Journal of the Neurological SciencesCitation Excerpt :Multiple prior studies were however limited by absence of an age-matched control population, and the reported prevalence of 15–63% is likely exaggerated [156,161]. Nevertheless, white matter lesions in LNB have been described with increased prevalence, including in the pediatric population, and likely do reflect part of the imaging spectrum of LNB (Fig. 10) [157,162]. Besides the white matter lesions, parenchymal brain involvement in LNB has been described in isolated case reports and does not have any specific involvement pattern.
Congenital and Acquired Facial Palsies
2015, Neuromuscular Disorders of Infancy, Childhood, and Adolescence: A Clinician's ApproachParainfectious meningo-encephalo-radiculo-myelitis (cat scratch disease, Lyme borreliosis, brucellosis, botulism, legionellosis, pertussis, mycoplasma)
2013, Handbook of Clinical NeurologyCitation Excerpt :Bb-specific antigens, specific IgM and IgG immune complexes, and positive PCR and culture are also reported. Computed tomography is usually normal; Magnetic resonance imaging (MRI) can show discrete or confluent white matter abnormalities on T2-weighted images, which may or may not enhance and often resemble MRI findings in ADEM (Fig. 125.1) (Belman et al., 1992; Wilke et al., 2000). The goal of LB therapy is to eradicate the organism.
Imaging of Rickettsial, Spirochetal, and Parasitic Infections
2012, Neuroimaging Clinics of North AmericaCitation Excerpt :MR imaging findings of the brain in Lyme disease are often within normal limits, even in patients with known Lyme disease who have neurologic manifestations.14,28 There are several studies in the literature that have evaluated MR neuroimaging findings in patients with Lyme disease and identified multiple bilateral periventricular and/or subcortical foci of T2 prolongation in patients with neurologic symptoms.32,63–67 Some of the lesions are said to resemble multiple sclerosis plaques with involvement of the callososeptal interface (Fig. 8).31,33
The Sleep-Immunity Relationship
2012, Neurologic ClinicsFacial and Bulbar Weakness
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Presented in part at the 43rd Annual Meeting of the American Academy of Neurology, April, 1991, Boston.
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Supported in part by a grant for Lyme Disease Research from the State of New York and in part by PHS RO1 AR40470 (PKC, ALB).