Brief reportsSusac syndrome
Section snippets
Acknowledgements
The authors acknowledge Drs. Kazuhiro Tashima, Eiichiro Uyama, Makoto Uchino (Department of Neurology, Kumamoto University School of Medicine), Youichiro Hashimoto (Department of Neurology, Kumamoto City Hospital), and Toshiro Yonehara (Department of Neurology, Stroke Center, Saiseikai Kumamoto Hospital) for helpful clinical cooperations.
References (5)
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Microangiopathy of the brain, retina, and cochlea (Susac syndrome). A report of five cases and a review of the literature
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Microangiopathy of the brain and retina
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Cited by (30)
Susac's syndrome: A case of simultaneous development of all three components of the triad
2013, Journal of the Neurological SciencesCitation Excerpt :Moreover, the encephalopathy may prevent patients from complaining of visual and hearing difficulty. Peripherally located branch retinal artery occlusions may not result in visual symptoms, and may only be recognized by an ophthalmologist [13–15]. Thus, MR imaging is often necessary to establish the diagnosis.
Susac syndrome: Variable onset modes and disease courses: Two case reports
2010, Journal Francais d'OphtalmologieRetinal Infarcts in a Patient with an Acute Confusional Syndrome
2009, Survey of OphthalmologyCitation Excerpt :Susac syndrome is a rare disease of unknown pathogenesis. It is characterized by an arteriolar microangiopathy that affects brain, retina, and cochlea, producing encephalitis, retinal infarcts, and neurosensory hearing loss.3,7 This disease is commonly underdiagnosed9,11 and requires a high index of suspicion to make the correct diagnosis.
Central retinal artery occlusion as the initial ophthalmic presentation of Susac's syndrome
2004, Canadian Journal of OphthalmologyDisorders of cochlear blood flow
2003, Brain Research Reviews