Causes of acute renal dysfunction after percutaneous coronary intervention and comparison of late mortality rates with postprocedure rise of creatine kinase-MB versus rise of serum creatinine
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Cited by (55)
Predicting Contrast-induced Renal Complications
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2020, Interventional Cardiology ClinicsUsefulness of the SYNTAX score to predict acute kidney injury after percutaneous coronary intervention (from the acute catheterization and urgent intervention triage strategy trial)
2014, American Journal of CardiologyCitation Excerpt :There are several potential explanations for this finding. First, patients with more complex coronary disease typically require more significant and challenging interventions in the catheterization lab, and it has been previously shown that complex PCI is associated with greater risk for postprocedural contrast nephropathy.21 In our findings from ACUITY, this is suggested by the fact that both the number of vessels treated and procedure time increased significantly with SS.
Vasoactive and antiarrhythmic drugs during percutaneous coronary intervention
2013, Interventional Cardiology ClinicsCitation Excerpt :However, CI-AKI has been reported to occur in up to 50% of patients with diabetic nephropathy and a mean serum creatinine of 5.9 mg/dl.30 Several studies have reported an association between CI-AKI and an increased incidence of myocardial infarction, target vessel revascularization, and death after PCI.31,32 Currently, the mechanism causing CI-AKI is incompletely understood, but direct cytotoxicity of iodinated contrast agents and disturbances in renal hemodynamics have been identified to be contributing factors.33
This study was supported by the Cardiovascular Research Institute, Washington, DC.