Index by author
Abe, T.
- InterventionalYou have accessSuperior Petrosal Sinus: Hemodynamic Features in Normal and Cavernous Sinus Dural Arteriovenous FistulasR. Shimada, H. Kiyosue, S. Tanoue, H. Mori and T. AbeAmerican Journal of Neuroradiology March 2013, 34 (3) 609-615; DOI: https://doi.org/10.3174/ajnr.A3252
Agosta, F.
- BrainYou have accessThe Role of Amyloid-β, tau, and Apolipoprotein E ε4 in Alzheimer Disease: How Is the Team Playing?M. Filippi, E. Canu and F. AgostaAmerican Journal of Neuroradiology March 2013, 34 (3) 511-512; DOI: https://doi.org/10.3174/ajnr.A3295
Aisen, P.S.
- EDITOR'S CHOICEBrainOpen AccessApolipoprotein E ε4 Does Not Modulate Amyloid-β–Associated Neurodegeneration in Preclinical Alzheimer DiseaseR.S. Desikan, L.K. McEvoy, D. Holland, W.K. Thompson, J.B. Brewer, P.S. Aisen, O.A. Andreassen, B.T. Hyman, R.A. Sperling, A.M. Dale and for the Alzheimer's Disease Neuroimaging InitiativeAmerican Journal of Neuroradiology March 2013, 34 (3) 505-510; DOI: https://doi.org/10.3174/ajnr.A3267
These authors investigated the relationship between amyloid-β, phosphorylated tau, and apolipoprotein E in preclinical Alzheimer disease in 170 patients and controls who had serial imaging up to 3.5 years after the initial study. Global volumes as well as volumes in brain regions known to be involved in the chronic stages of the disease were assessed. Their results led them to propose that atrophy rates are primarily influenced by apolipoprotein E via amyloid-β mechanisms and that amyloid-β -associated neurodegeneration occurs only in the presence of phosphorylated tau.
Ambarki, K.
- BrainYou have accessAutomated Determination of Brain Parenchymal Fraction in Multiple SclerosisM. Vågberg, T. Lindqvist, K. Ambarki, J.B.M. Warntjes, P. Sundström, R. Birgander and A. SvenningssonAmerican Journal of Neuroradiology March 2013, 34 (3) 498-504; DOI: https://doi.org/10.3174/ajnr.A3262
Andreassen, O.A.
- EDITOR'S CHOICEBrainOpen AccessApolipoprotein E ε4 Does Not Modulate Amyloid-β–Associated Neurodegeneration in Preclinical Alzheimer DiseaseR.S. Desikan, L.K. McEvoy, D. Holland, W.K. Thompson, J.B. Brewer, P.S. Aisen, O.A. Andreassen, B.T. Hyman, R.A. Sperling, A.M. Dale and for the Alzheimer's Disease Neuroimaging InitiativeAmerican Journal of Neuroradiology March 2013, 34 (3) 505-510; DOI: https://doi.org/10.3174/ajnr.A3267
These authors investigated the relationship between amyloid-β, phosphorylated tau, and apolipoprotein E in preclinical Alzheimer disease in 170 patients and controls who had serial imaging up to 3.5 years after the initial study. Global volumes as well as volumes in brain regions known to be involved in the chronic stages of the disease were assessed. Their results led them to propose that atrophy rates are primarily influenced by apolipoprotein E via amyloid-β mechanisms and that amyloid-β -associated neurodegeneration occurs only in the presence of phosphorylated tau.
Apostol, M.A.
- SpineYou have accessCT-Guided Cervical Transforaminal Steroid Injections: Where Should the Needle Tip Be Located?J.K. Hoang, D.P. Massoglia, M.A. Apostol, C.D. Lascola, J.D. Eastwood and P.G. KranzAmerican Journal of Neuroradiology March 2013, 34 (3) 688-692; DOI: https://doi.org/10.3174/ajnr.A3266
Arquizan, C.
- FELLOWS' JOURNAL CLUBInterventionalYou have accessEvaluation of an Intravenous-Endovascular Strategy in Patients with Acute Proximal Middle Cerebral Artery OcclusionJ.-F. Vendrell, R. Mernes, N. Nagot, D. Milhaud, K. Lobotesis, V. Costalat, P. Machi, I.L. Maldonado, C. Riquelme, C. Arquizan and A. BonafeAmerican Journal of Neuroradiology March 2013, 34 (3) 603-608; DOI: https://doi.org/10.3174/ajnr.A3230
Here, the safety and efficacy of mechanical thrombectomy after intravenous therapy failure were assessed in 123 patients with acute MCA occlusions. All patients imaged were within 4.5 hours of onset, had DWI ASPECTS greater than 5, and variable NIHSS scores (8–25). The authors found that mechanical thrombectomy after failure of intravenous thrombolysis improves clinical outcomes at 3 months and could represent an alternative in the management of patients with acute MCA occlusion. Additionally, no symptomatic intracranial hemorrhages were detected in patients treated this way, suggesting this protocol is safe.